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The picture above is of a severely dysplastic
hip joint as would be seen on radiograph. The
shape of the head is no longer smooth and round,
there is no definition to the "neck"
which has become filled in and thickened basically
trying to form a new wider surface area to support
the body weight of the dog.
The "socket" is shallow and does not
wrap well around the head (at least >50%) so
there is instability and the bad fit leads to
arthritic changes due to the friction between
the bones and cartilage |
What is Hip Dysplasia?
Hip Dysplasia
is defined as a deformity of the Coxofemoral (hip) joint
that occurs during the growth period. Hip Dysplasia
is a hereditary condition that creates a poorly fitting
hip joint. As the dog walks on this joint, arthritis
will eventually develop, causing pain in the joint.
The degree of lameness that occurs is usually dependent
upon the extent of arthritic changes in the hip joint.
Is
this found in certain breeds of dogs?
Most breeds of
dogs can be affected with hip Dysplasia although it
is predominantly seen in the larger breeds of dogs,
such as the German Shepherd, St. Bernard, Labrador Retriever,
Pointers, and Setters. There is equal distribution of
the disease between male and female dogs. Unfortunately
Epagneuls Bretons are not free of the problem either,
but their smaller size often allows the condition to
go undetected, and why so many that are affected can
lead a relatively normal life.
What
are the clinical signs, and when do they occur?
The typical clinical
signs of hip Dysplasia are rear leg pain, in-coordination,
restriction in the movement and a reluctance to rise.
Wasting of the large muscle groups in the rear limbs
may eventually develop. Most owners report that the
dog has had difficulty in getting up from a lying position
for a period of weeks or months; lameness and pain subsequently
develop. Again, the severity of signs and progression
of the disease usually correlate with the extent of
arthritis in the joint. There are two major groups
of dogs that are presented to a veterinarian for dysplastic
changes, even though Clinical signs can occur as early
as 4-6 weeks of age :
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Dogs four to eight
months old, where genetic and environmental influences
have combined to overpower the normal remodeling
capacity of the hip. |
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Dogs one to ten years
old, where development of degenerative joint disease
is milder and slower. |
The young dogs
often have difficulty rising (especially in the morning),
and may show gait problems when walking, running or
using stairs. Their pain comes from many sources; tiny
fractures where the loose femur hits the rim of the
acetabulum, torn muscles, and torn joint capsule.
SIGNS OF CHD
IN YOUNG DOGS: What you will see first is a pup
that runs with both back legs nearly together, almost
like a rabbit would run. After exercise the pup will
be reluctant to rise, will sit back as if unsteady and
will have difficulty climbing stairs or inclines. The
pup might look slightly underdeveloped in the rear quarters.
When it stands the rear legs may not be parallel, but
rather too near each other at the hocks (ankles) called
"cow hocked".
You might notice a boniness to the pelvic area from
lack of good muscle development. Another hint of trouble
is an inability to extend the leg backward very far
(decreased range of motion). Note: Many pups rest or
sleep in a frog-like position with knees extended out
to either side - this is a good sign and shouldn't alarm
you.
In severe cases of Dysplasia, the young dog will rock
forward to support more weight on the front legs (which
can create trouble in the shoulders and elbows). When
dogs do this it seems as if they are tip-toeing or walking
very lightly on their rear legs. A dysplastic pup will
be reluctant to jump or "stand up" on its
hind legs. Signs usually being between five and eights
months of age. But remember, some dogs do not show any
signs at all of hip joint degeneration until mature
adults.
How
is it diagnosed?
Tentative diagnosis
of hip Dysplasia is made on the basis of history, breed,
and clinical signs. A large breed dog that
has been slow to rise for several months and now is
lame is highly suspect for hip Dysplasia; a dog which
refuses to rise should also be considered a candidate.
Because the clinical signs may mimic other diseases,
final diagnosis of hip Dysplasia can only be made
on the basis of specific radiographic (x-ray) findings.
To obtain the proper radiographs, dogs must be carefully
positioned on the radiographic table. This procedure
may require the use of a short-acting anesthetic or
sedation. The radiographs are evaluated for abnormal
shape of the hip joint and for degenerative changes
(arthritis).
How
is it treated?
The degree of
clinical signs and arthritic changes in the joints determine
the specific approach to therapy. Treatment of hip Dysplasia
may involve the use of drugs or surgery, or both. The
options are as follows:
l. Anti-inflammatory
drugs. Several drugs will give relief from pain. Aspirin
or acetaminophen may work well in some dogs. Other
steroidal (cortisone) and non-steroidal drugs may
also be used. Most have some side-effects and most
require administration once or twice daily. Many dogs
have severe stomach irritation to ibuprofen, so this
drug is not recommended. Unfortunately, it is not
possible to predict which dog will respond to which
drug. Therefore, a series of trials may be needed
to find the most effective one for your dog. Extreme
caution is advised when these drugs are given to dogs
with a history of kidney disease or with marginal
kidney function. Many of these drugs have an adverse
effect on blood flow to the kidneys and can lead to
kidney failure. This does not appear to be a concern
if kidney function is normal. As alluded to above,
dogs with a history of ulcers are also at risk for
complications. Your veterinarian can determine the
risk for your dog. Anti-inflammatory drug therapy
is most often used in older dogs, in dogs that did
not get good relief from surgery, or in dogs for which
surgery is not feasible.
There has been
some good pain relief achieved with a period of using
Adequan injectable for cartilage preservation, and
regeneration, with Canine Hip Dysplasia and other
arthritis conditions of joints. To this same
end many have reported good results with oral supplementations
such as Synoflex.
2. Surgery:
There are four main procedures: pectineal myotomy
(muscle cutting surgery), femoral head ostectomy
(ball removal), triple osteotomy, and hip
joint replacement. Pectineal myotomy is
a relatively minor procedure that involves cutting
a small muscle that puts pressure on the hip joint.
It results in no loss of leg function and gives good
to excellent relief in 80-90% of dogs. If both hips
are abnormal, both hips may be operated on at the
same time. The dog recovers from surgery in one to
two days. However, this procedure does not stabilize
the hip joint or prevent progression of arthritic
changes. Within a few months to several years, pain
and lameness will return. This procedure is especially
recommended in older dogs. Femoral head ostectomy
(FHO) is another choice. The hip joint is a ball
and socket joint. FHO is the removal of the ball part
of the joint. This gives excellent results in small
dogs because a functional "false joint"
forms. However, some large dogs may not form this
"false joint" very well. This procedure
is usually used in large dogs if arthritis is very
severe, if the hip dislocates, or if the expense of
the other procedures is prohibitive. Triple osteotomy
is a procedure in which the pelvis is cut in three
places around the hip joint. The bone is rotated to
create better alignment with the femoral head (the
ball). It is reattached so that the joint functions
in a more normal fashion without looseness and pain.
This should only be performed in a dog with no arthritic
changes in the joint, and most successful when done
in very young dogs if detected early. It is an expensive
procedure. Hip joint replacement is possible,
as is done in humans. A stainless steel ball and socket
are attached to the pelvis and femur in place of the
abnormal ones. It is another expensive procedure,
but it may give many years of pain-free use of the
hips. Although the intent is for the transplant to
be permanent, the new joint may loosen after a period
of time.
I
am considering breeding my dog. Can anything be done
toprevent hip dysplasia in the puppies?
Research has
shown that the cause of hip Dysplasia is related to
a combination of genetic and environmental factors,
the term "polygenic" is used. The
disease is known to be an inherited condition and
the genetics of hip Dysplasia are extremely complicated.
In addition, environmental factors such as overfeeding,
poor nutrition and excessive exercise can predispose
a dog (especially growing puppies) to developing hip
Dysplasia if they already have the genetic makeup
for the disease. Because the inheritance of the disease
is so complicated, many questions remain regarding
eradication of the disease. Here are some practical
suggestions:
1. Have your
dog radiographed before breeding to be sure the hips
are normal. If they are not, this dog should not be
bred.
2. Consider
a feeding program to keep growth to a moderate rate.
There is a growing body of evidence indicatingthat
dogs that grow very rapidly are more likely to have
hip Dysplasia. Many authorities recommend feeding
an adult-type food to puppies of high risk breeds
so their growth is slower. They will still reach their
full genetic body size, but just not as rapidly. Some
dog food manufacturers are now making puppy foods
for large breed dogs. This is essentially the same
approach as feeding an adult food because these puppy
foods are formulated for slower growth.
3. Avoid excessive
exercise in a growing puppy. Any abnormality in the
structure of the hip joint is magnified if excessive
running and jumping occur. It is not necessary to
treat your puppy as it were handicapped, but long
sessions of running or chasing thrown objects can
be detrimental to joints genetically predisposed to
Hip Dysplasia. Do not expect your puppy to hunt
for the same periods as and adult dog, especially
over rough terrain when not fully mature.
4. All
puppies are born with normal hip joints. It
is around 60 days of life that the normal joint starts
to develop abnormally. Good traction in the
whelping box may prevent excessive wear and tear early
on, and good traction thereafter can do no harm as
they are going through the rapid growth phase up to
about 5 months of age. Most Epagneul Bretons reach
full growth at 18 months of age.
What
does it mean to have the hips certified as normal?
The Orthopedic
Foundation for Animals (O.F.A.) is an organization
established for the purpose of standardizing the evaluation
process of canine hip radiographs. The O.F.A. consists
of a board of certified veterinary radiologists who
are skilled in detecting hip Dysplasia. If the radiographs
submitted to the O.F.A. are declared normal, the dog
is issued an O.F.A. certificate number indicating
that it has normal hip confirmation. The O.F.A. requires
that dogs must be a minimum of two years of age to
be certified. Many dedicated breeders require that
a dog must have an O.F.A. certificate before breeding
is allowed. Another hip evaluation program is called
the PennHip method. Radiographs are made of the anesthetized
dog in such a manner as to place outward force on
the hip joints. This can reveal looseness in the joints
that may elude detection by the more standard radiographic
methods. It is also useful in identifying hip Dysplasia
in puppies as young as four months of age. Although
any veterinarian can make the appropriate radiographs
and submit them for O.F.A. certification, the PennHip
method must be performed by a veterinarian specifically
trained and certified in this procedure. There
is considerable scientific research about Canine Hip
Dysplasia and anyone can find just about any documentation
on which method is better than another, but it still
remains that the only real way to identify the condition
is with some form of radiograph.
How
does OFA know that the hip radiographs belong to my
dog?
The radiographs
must be imprinted with identification information
about your dog at the time they are made and developed.
This procedure creates a permanent mark on the radiograph.
In addition, OFA now requires that certified dogs
be permanently marked with either a tattoo or a microchip
implant, or have a DNA profile on file. The microchip
implant process is simple and very effective. A tiny
microchip is implanted under your dog's skin through
a special injection needle. A special scanner can
detect these chips through the skin. They can identify
the dog and its owner through its code number and
a registry system. This is also an excellent means
of getting lost dogs back home because the registry
system is national in scope. Most dogs imported from
European countries are also permanently identified
with a tattoo and this number is also often recorded
on the radiograph as well.
How
are the ratings defined and what does it mean to me?
|
OFA
RATING (min. Age 2yr.) |
|
SCC
Rating |
|
(min.
1 yr & Larger 18 mos) |
|
FCI
SYSTEM |
British Veterinary Association System |
|
Excellent
* |
A
|
A1
|
Score 0-4
(None >3/hip) |
|
Good
* |
A
|
A2
|
Score
5-10 (None >6/hip) |
|
Fair
* |
B
|
B
1-2 |
Score 11-18 |
|
Indeterminate
* |
B
|
C1
|
Score 19-25 |
|
Mild
|
C
|
C2
|
Score 26-35 |
|
Moderate
|
D
|
D
|
Score 36-50 |
|
Severe
|
E
|
E
|
Score 51-106 |
Categories
in blue boxes under OFA are issued certificates that
hips are within normal limits, those also listed with
and asterisk are acceptable for CEB-USA ratings at this
time; categories under SCC in blue boxes are those that
are considered acceptable for CEB-France breed title
& ratings, not all breeders follow the recommended
guidelines. Most all systems agree on the Excellent,
Good, Moderate, and Severe categories, There is
a fine line between Indeterminate (no arthritic changes)
and Fair (joint is not perfect but shows no degeneration)
and is often a judgment call by the radiologist, and
is one reason OFA elects to recheck this category 6
months later often calling it "borderline".
Understanding
the ratings will help breeders make informed decisions
with regards to breeding partners. Not all systems
are equal to each other, the intent is the same to find
those affected. Evaluating pedigrees of prospective
stock is done on 2 levels:
1.
Pedigree depth - how many generations of normal dogs
are found in a pedigree. OFA suggests that pedigrees
with more generations of normal dogs lessens the risk
of the new generation having affected. It is no
guarantee that there will be no affected but offers
some hope. A 3 generation pedigree of all clears
has shown significant risk reduction, according to OFA.
2.
Pedigree breadth - the number of related dogs to those
listed in the pedigree are also normal. Knowing
the littermate information of the dogs listed in the
pedigree tells you how consistent a pedigree is for
passing on sound hips. Unfortunately in the early
days in the USA, this information was unavailable, but
now with the posting of litter number registration you
can track dogs with the same litter number under OFA.
You can also track information if provided on the newer
OFA forms by Sire or Dam registration numbers as well
to give you this breadth information on production.
3.
A system is only really valuable if all information
is submitted both normal and affected. Sadly in
the USA the OFA charges for both normal and abnormal
submissions and because of this most affected are not
reported. Dedicated breeders submit affected as
well to make a true tracking of genetic information
available for all to succeed. We urge all to submit
every evaluation to the OFA and to have it listed in
the Open Registry format. It is not a crime to
have an affected dog and it does not mean that an entire
family of dogs should be condemned if an individual
is found to have a problem, the information can benefit
all breeders in retaining individuals within a line
that appear to reproduce better than themselves, even
if it means starting with affected stock.
4.
When investigating potential purchases inquire as to
whether or not hip evaluation has been done on parents
and then grand parents. If the breeder cannot
provide you with parent information and a registration
number to investigate on the OFA web site then you would
have to assume there is no rating or worse that the
dog is affected. In many Epagneul Breton pedigrees
in the USA there is very limiting information on hips
available as many early breeders did not do evaluations
erroneously believing that the breed had no such problems.
American Brittany breeders took advantage of OFA services
when they were started in the 1960's and have made great
strides in their stock and have some valuable data for
their gene pool. We have a long way to go with
the Epagneul Breton but hopefully we will be as successful
in our attempts.
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